The formulation of Serratiopeptidase, Paracetamol and Diclofenac potassium is introduced in India with the brand name of FULDASE. This combination is a potent analgesic, antipyretic, anti-edematous and anti-inflammatory.
Serratiopeptidase is a proteolytic enzyme, which binds to alpha-2-macroglobulin in the blood, there by, helps to mask us agnogenicity but retains its enzymatic activity and is slowly transferred to site of inflammation. It hydrolyses bradykinin, histamine and serotonin responsible for oedematis status. It reduces swelling and edema, improves microcirculation and accelerates the elimination of sputum by breaking down the liquifying mucus secretions and fibrin clots, hence it acts as antiinflammatory, antiedematous and anti-fibrinolytic. One of the useful actions of it is that it increases the concentration of antibiotic at the site of infection.
Paracetamol is a non-steroidal antiinflammatory drug and is a para-aminophenol derivative. It is centrally acting analgesic and a potent antipyretic, but devoid of a significant antiinflammatory effect which could be due to its weak activity on peripheral prostaglandin synthetase. This drug does not produce acid-base imbalance, electrolyte disturbances, and gastrointestinal irritation nor does it effect blood clotting factors. It also posses uricosuric property to varying degree and are non-addicting. Paracetamol is rapidly absorbed on oral administration. Peak plasma levels are reached within 1/2 to 1-hour. It is mainly excreted in urine as conjugation products of glucuronic and sulfuric acids. The ability of infant’s liver for glucuronidation of paracetamol is poor.
Diclofenac potassium is anonsteroictal, antiinflammatory, analgesic and antipyretic drug, which inhibits prostaglandin synthesis by interfering with the action of prostaglandin synthetase. It is ideally suited for patients on sodium free diet, hypertensive patients and those on diuretic therapy.
Paracetamol enhances oral anticoagulant activity. Its absorption is increased by meloclopramide and reduced by pethidine and propantheline. Chronic use of alcohol potentiates hepatotoxicity by paracetamol.
No drug interaction regarding Serratiopeptidase and diclofenac potassium is reported.